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07-SEPTEMBER-2008 03:17:44 - Dyslexia Part of a series on Dyslexia and related disorders Education · Neuropsychology RELATED CONDITIONS Alexia Auditory Processing Disorder Dyscalculia · Dysgraphia Dyslexia · Dyspraxia THEORIES Double deficit · Magnocellular Perceptual noise exclusion Phonological deficit RELATED TOPICS IDEA · Literacy Reading acquisition · Spelling Recording for the Blind Dyslexic LISTS Assessments · Fields People · Publications Topics Fiction · Treatments view talk This article is about developmental dyslexia. For acquired dyslexia, see Alexia disorder. Dyslexia is considered to be a learning disability. It manifests primarily as a difficulty with written language, particularly with reading and spelling. It is separate and distinct from reading difficulties resulting from other causes, such as a non-neurological deficiency with vision or hearing, or from poor or inadequate reading instruction.1 Evidence also suggests that dyslexia results from differences in how the brain processes written and/or spoken language. Although dyslexia is allegedly the result of a neurological difference, it is not an intellectual disability. Dyslexia occurs at all levels of intelligence.2 The word dyslexia comes from the Greek words δυσ- dys- impaired and λÎξις lexis word. People with dyslexia are called dyslexic. Contents 1 Overview 2 History 3 Subtypes of developmental dyslexia 4 Variations and related conditions 5 Scientific research 5.1 Theories of developmental dyslexia 5.1.1 The evolutionary hypothesis 5.1.2 The phonological hypothesis 5.1.3 The rapid auditory processing theory 5.1.4 The visual theory 5.1.5 The cerebellar theory 5.1.6 The magnocellular theory 5.1.7 Perceptual visual-noise exclusion hypothesis 5.2 Research using functional brain scan technology 5.3 Research also indicates 5.3.1 Genetic factors 5.3.2 Physiology 5.4 Effect of language orthography 6 Characteristics 6.1 Speech, hearing and listening 6.2 Reading and spelling 6.3 Writing and motor skills 6.4 Mathematical abilities 6.5 Creativity, arts, business 7 Management 8 Facts and statistics 9 Legal and educational support issues 10 Controversy 11 Dyslexia in literature, film, and television 12 See also 13 References 14 External links 14.1 Historical 14.2 Research papers, articles and media 14.3 Regional associations and organizations 14.4 Support groups and organizations 14.5 Dyslexia, Learning Disabilities, and Special Education Information 14.6 Dyslexic People Overview Dyslexia Classification and external resources ICD-10 F81.0, R48.0 ICD-9 315.02, 784.61 OMIM 127700 604254 606896 606616 608995 300509 DiseasesDB 4016 MeSH D004410 Dyslexia is most commonly characterized by difficulties with learning how to decode words, to spell, and to read accurately and fluently. Dyslexia also makes mathematics more difficult as people with dyslexia might mix numbers up. Dyslexic individuals often have difficulty breaking the code of sound-letter association the alphabetic principle, and they may also reverse or transpose letters when writing or confuse letters such as b, d, p, q, especially in childhood. However, dyslexia is not a visual problem that involves reading letters or words backwards or upside down, nor are such reversals a defining characteristic of dyslexia. Many individuals with dyslexic symptoms involving reading, writing, and spelling also exhibit symptoms in other domains such as poor short-term memory skills, poor personal organizational skills and problems processing spoken language. 3 These symptoms may coexist with or overlap with characteristics among others of attention-deficit hyperactivity disorder, auditory processing disorder, 4 developmental dyspraxia, dyscalculia, and/or dysgraphia. However, dyslexia and attention deficit/hyperactivity disorder are not correlated developmental problems.5 Evidence that dyslexia is of neurological origin is substantial. Acquired dyslexias occur due to brain damage in the left hemisphere's key language areas, which may be a clue when it comes to developmental types. Research also suggests an association with biochemical and genetic markers.678 However, experts disagree over the precise definition and criteria for diagnosis, and some advocate that the term dyslexia be dropped altogether and replaced with the term reading disorder or reading disability RD. Because reading skills occur on a continuum with no clear distinction between typical readers and dyslexic readers, some experts assert that the term dyslexia should be reserved for the two to five percent with the most severe reading deficits. 9 Dyslexia is a lifelong condition that cannot be reversed, but appropriate remedial instruction and compensatory strategies can help dyslexic individuals mitigate or overcome their difficulties with written language. 10 A large body of evidence shows which types of instruction dyslexics need to be successful. 11 Many dyslexics overcome early problems with literacy and go on to earn advanced degrees and pursue successful careers; a high level of motivation coupled with strong encouragement and mentorship have been identified as factors leading to their success. 12 History Identified for the first time by Oswald Berkhan in 1881, 13 the term 'dyslexia' was coined in 1887 by Rudolf Berlin, an ophthalmologist practicing in Stuttgart, Germany.14 He used the term to refer to a case of a young boy who had a severe impairment in learning to read and write in spite of showing typical intellectual and physical abilities in all other respects. In 1896, W. Pringle Morgan, a British physician, from Seaford, East Sussex, England published a description of a reading-specific learning disorder in a report to the British Medical Journal titled Congenital Word Blindness. This described the case of a 14-year-old boy who had not yet learned to read, yet showed normal intelligence and was generally adept at other activities typical of children of that age.15 During the 1890s and early 1900s, James Hinshelwood, a British ophthalmologist, published a series of articles in medical journals describing similar cases of congenital word blindness, which he defined as a congenital defect occurring in children with otherwise normal and undamaged brains characterised by a difficulty in learning to read. In his 1917 book Congenital Word Blindness, Hinshelwood asserted that the primary disability was in visual memory for words and letters, and described symptoms including letter reversals, and difficulties with spelling and reading comprehension.16 A key early researcher in dyslexia was Samuel T. Orton, a neurologist who worked primarily with stroke victims. In 1925 Orton met a boy who could not read and who exhibited symptoms similar to stroke victims who had lost the ability to read. Orton began studying reading difficulties and determined that there was a syndrome unrelated to brain damage that made learning to read difficult. Orton called the condition strephosymbolia meaning 'twisted signs' to describe his theory that individuals with dyslexia had difficulty associating the visual forms of words with their spoken forms.17 Orton observed that reading deficits in dyslexia did not seem to stem from strictly visual deficits.18 He believed the condition was caused by the failure to establish hemispheric dominance in the brain.19 He also observed that the children he worked with were disproportionately left- or mixed-handed, although this finding has been difficult to replicate.20 Orton's hypothesis concerning hemispheric specialization was borne out by post-mortem studies in the 1980s and 1990s establishing that the left planum temporale, a brain area associated with language processing, is physically larger than the corresponding right area in the brains of non-dyslexic subjects, but that these brain areas are symmetrical or slightly larger on the right for dyslexic subjects.21 FMRI imaging studies of children and young adults reported in 2003 provide further support, demonstrating that increases in age and reading level are associated with a suppression of right hemispheric activity.22 23 Influenced by the kinesthetic work of Helen Keller and Grace Fernald, and looking for a way to teach reading using both left and right brain functions,24 Orton later worked with psychologist and educator Anna Gillingham to develop an educational intervention that pioneered the use of simultaneous multisensory instruction. The Orton-Gillingham approach to remedial reading instruction is still widely used and forms the basis of many reading intervention programs. 25 In contrast, Dearborn, Gates, Bennet and Blau considered a faulty guidance of the seeing mechanism to be the cause. The data collected in 1931 by Tinker and Goodenough The J.Educ. Psych.26 seemed to support this thesis. They wanted to know if a conflict between spontaneous orientation of the scanning action of the eyes from right to left and training aimed at the acquisition of an opposite direction would allow an interpretation of the facts observed in the dyslexic disorder and especially of the ability to mirror-read. To this end the authors asked four adults to read a text reflected in a mirror for ten minutes a day for a period of five months. It was confirmed that in all the subjects, the words were not perceived in their globality but required a meticulous analysis of the letters and syllables. Moreover they also demonstrated either total or partial inversions even sometimes affecting the order of the words in a sentence. What is more, they revealed a curious impression of not just horizontal but also vertical inversions.These are errors that exist amongst dyslexics and they suffer from the aggravating circumstance inherent in all learning. What remained to be demonstrated was that there exists a preference amongst dyslexics, without sensory deficiency, or mental retardation, or any backwardness in speech or language, towards scanning with the eyes from right to left. Proof of this was provided in a work conducted under Clement Launay in 1949 thesis G. Mahec Paris 1951. In adult subjects the reading of a series of 66 tiny lower-case letters, 5 mm high, spaced 5 mm apart, first from left to right and then from right to left was more easily and quickly done in the left to right direction. For former dyslexic children, a substantial number read a series of 42 letters with equal speed in both directions and some 10% read better from right to left than from left to right. The phenomenon is clearly linked to the dynamics of sight as it disappears when the space between letters is increased, transforming the reading into spelling. This experience also explains the ability to mirror-read. This reading test can also be used to diagnose serious cases of dyslexia. In the 1970s, a new hypothesis, based in part on Orton's theories, emerged that dyslexia stems from a deficit in phonological processing or difficulty in recognizing that spoken words are formed by discrete phonemes for example, that the word CAT comes from the sounds k, æ, and t. As a result, affected individuals have difficulty associating these sounds with the visual letters that make up written words. Key studies of the phonological deficit hypothesis include the finding that the strongest predictor of reading success in school age children is phonological awareness,26 and that phonological awareness instruction can improve decoding skills for children with reading difficulties.27 The advent of neuroimaging techniques to study brain structure and function enhanced the research in the 1980s and 1990s. Since then, interest in the neurologically based causes has persisted. Current models of the relation between the brain and dyslexia generally focus on some form of defective or delayed brain maturation. More recently, genetic research has provided increasing evidence supporting a genetic origin of dyslexia 28. Researchers are currently searching for a link between the neurological and genetic findings, and the reading disorder. There are many previous and current theories of dyslexia, but the one that has the most support from research is that, whatever the biological cause, dyslexia is a matter of reduced phonogical awareness, the ability to analyze and link the units of spoken and written languages. 29. Subtypes of developmental dyslexia Studies by Castles and Coltheart suggest that developmental dyslexia includes at least two prevalent and distinct varieties or subtypes of dyslexia. Subtypes include surface dyslexia and phonological dyslexia. Understanding these subtypes is useful in diagnosing learning patterns and developing approaches for overcoming impairments that may be visual perception impairments or speech discrimination deficits. These subtypes are based on differing patterns of underlying symptoms, as supported by a finding using large-scale data from comparative studies of reading patterns in dyslexic and normal readers 30. In the study by Castles and Coltheart, 56 dyslexic boys and 56 non-dyslexic boys as a control group were tested. During the test, the boys read aloud words and non words that were presented to them. The researchers found that surface dyslexics subjects who have poor lexical skills, or can't make out irregular words well had a mean difference of 14.4 words between reading regular words versus irregular words, however, the mean difference in subjects with phonological dyslexia subjects who can't use sub lexical skills, or can't make out non words was only 7.75 words which was comparable to the control group 30. Surface dyslexia is the outcome of an individual who cannot function using the lexical procedure for reading out loud. The lexical procedure includes sounding out a word though the use of a past word already known 30. Phonological dyslexia Phonological dyslexia is characterized by subjects who can read aloud both regular and irregular words but have difficulties with non words and with connecting sounds to symbols, or with sounding out words 30. Phonological processing tasks predict reading accuracy and comprehension. This subtype is the most predominant form of dyslexia 31 Castles, A., Coltheart, M. 1993. Varieties of developmental dyslexia. Cognition, 472, 149-180 /ref.In Castles and Coltheart's study, they had 56 dyslexic boys and 56 non dyslexic boys read words and non words given to them. The majority of the boys, 55%, showed a phonological dyslexic pattern 30.In Castles and Coltheart's study, dyslexic subjects and control subjects were asked to read non words listed on a card, 17 out of 51 cases of dyslexics were below the confidence limit in non word reading, which was derived by the control group of subjects their own age. These phonological dyslexics have a lower non word reading level than expected by reviewing their irregular word reading level 32. Phonological dyslexia is the outcome of a subject who cannot function using the sub lexical pronunciations are constructed from smaller orthographic components procedure for reading out loud 30. In their study, Wolf and Bowers tested out naming speed by having their subjects name a symbol as quickly as possible when shown on a flash card. Difficulties in naming speed exist in conjunction with a phonological deficit, is characterized as double deficit dyslexia 33. Variations and related conditions Dyslexia is a learning disability. It has many underlying causes that are believed to be a brain-based condition that influences the ability to read written language. It is identified in individuals who fail to learn to read in the absence of a verbal or nonverbal intellectual impairment, sensory deficit e.g., a visual deficit or hearing loss, pervasive developmental deficit or a frank neurological impairment. The following conditions may also be contributory or overlapping factors, or underlying cause of the dyslexic symptoms as they can lead to difficulty reading: Auditory processing disorder is a condition that affects the ability to encode auditory information. It can lead to problems with auditory working memory and auditory sequencing. Many dyslexics have auditory processing problems including history of auditory reversals. Auditory processing disorder is recognized as one of the major causes of dyslexia. Cluttering is a speech fluency disorder involving both the rate and rhythm of speech, and resulting in impaired speech intelligibility. Speech is erratic and dysrhythmic, consisting of rapid and jerky spurts that usually involve faulty phrasing. The personality of the clutterer bears striking resemblance to the personalities of those with learning disabilities.34 Dyspraxia is a neurological condition characterized by a marked difficulty in carrying out routine tasks involving balance, fine-motor control, and kinesthetic coordination. Problems with short term memory and organization are typical of dyspraxics. This is most common in dyslexics who also have attention deficit disorder. Verbal dyspraxia is a neurological condition characterized by marked difficulty in the use of speech sounds, which is the result of an immaturity in the speech production area of the brain. Dysgraphia is a disorder which expresses itself primarily during writing or typing, although in some cases it may also affect eye-hand coordination in such direction or sequence oriented processes as tying knots or carrying out a repetitive task. Dysgraphia is distinct from Dyspraxia in that the person may have the word to be written or the proper order of steps in mind clearly, but carries the sequence out in the wrong order. Dyscalculia is a neurological condition characterized by a problem with learning fundamentals and one or more of the basic numerical skills. Often people with this condition can understand very complex mathematical concepts and principles but have difficulty processing formulas and even basic addition and subtraction. Scotopic sensitivity syndrome, also known as Irlen Syndrome, is a term used to describe sensitivity to certain wavelengths of light which interfere with proper visual processing. See also Orthoscopics and asfedia. Scientific research Theories of developmental dyslexia The following theories should not be viewed as competing, but viewed as theories trying to explain the underlying causes of a similar set of symptoms from a variety of research perspectives and backgrounds. The evolutionary hypothesis This theory posits that reading is an unnatural act, and carried out by humans for an exceedingly brief period in our evolutionary history Dalby, 1986. It has been less than a hundred years that most western societies promoted reading by the mass population and therefore the forces that shape our behavior have been weak. Many areas of the world still do not have access to reading for the majority of the population. There is no evidence that pathology underlies dyslexia but much evidence for cerebral variation or differences. It is these essential differences that are taxed with the artificial task of reading. 35 The native reading hypothesis of dyslexia is another evolutionary theory which argues that because spoken language is naturally learned in the first few years of development, similarly, written language is best learned at the same early age. It suggests that many forms of dyslexia are therefore, to some extent, a result of introducing reading too late in neurodevelopment. This means that the typically late reading of dyslexics might sometimes be the cause of dyslexia, rather than the other way around, and many cases of dyslexia might be prevented by the earlier introduction of reading instruction.36 The phonological hypothesis The phonological hypothesis postulates that dyslexics have a specific impairment in the representation, storage and/or retrieval of speech sounds. It explains dyslexics' reading impairment on the basis that learning to read an alphabetic system requires learning the grapheme/phoneme correspondence, i.e. the correspondence between letters and constituent sounds of speech. If these sounds are poorly represented, stored or retrieved, the learning of grapheme/phoneme correspondences, the foundation of reading by phonic methods for alphabetic systems, will be affected accordingly.37 The rapid auditory processing theory The rapid auditory processing theory is an alternative to the phonological deficit theory, which specifies that the primary deficit lies in the perception of short or rapidly varying sounds. Support for this theory arises from evidence that dyslexics show poor performance on a number of auditory tasks, including frequency discrimination and temporal order judgment. Backward masking tasks, in particular, demonstrate a 100-fold 40 dB difference in sensitivity between normals and dyslexics. 38 Abnormal neurophysiological responses to various auditory stimuli have also been demonstrated. The failure to correctly represent short sounds and fast transitions would cause further difficulties in particular when such acoustic events are the cues to phonemic contrasts, as in /ba/ versus /da/. There is also evidence that dyslexics may have poorer categorical perception of certain contrasts.37 The visual theory The visual theory Lovegrove et al., 1980; Livingstone et al., 1991; Stein and Walsh, 1997 reflects another long standing tradition in the study of dyslexia, that of considering it as a visual impairment giving rise to difficulties with the processing of letters and words on a page of text. This may take the form of unstable binocular fixations, poor vergence, or increased visual crowding. The visual theory does not exclude a phonological deficit, but emphasizes a visual contribution to reading problems, at least in some dyslexic individuals. At the biological level, the proposed aetiology of the visual dysfunction is based on the division of the visual system into two distinct pathways that have different roles and properties: the magnocellular and parvocellular pathways. The theory postulates that the magnocellular pathway is selectively disrupted in certain dyslexic individuals, leading to deficiencies in visual processing, and, via the posterior parietal cortex, to abnormal binocular control and visuospatial attention. Evidence for magnocellular dysfunction comes from anatomical studies showing abnormalities of the magnocellular layers of the lateral geniculate nucleus Livingstone et al., 1991, psychophysical studies showing decreased sensitivity in the magnocellular range, i.e. low spatial frequencies and high temporal frequencies in dyslexics, and brain imaging studies.37 The cerebellar theory Yet another view is represented by the automaticity/ cerebellar theory of dyslexia. Here the biological claim is that the dyslexic's cerebellum is mildly dysfunctional and that a number of cognitive difficulties ensue. First, the cerebellum plays a role in motor control and therefore in speech articulation. It is postulated that retarded or dysfunctional articulation would lead to deficient phonological representations. Secondly, the cerebellum plays a role in the automatization of overlearned tasks, such as driving, typing and reading. A weak capacity to automatize would affect, among other things, the learning of grapheme±phoneme correspondences. Support for the cerebellar theory comes from evidence of poor performance of dyslexics in a large number of motor tasks, in dual tasks demonstrating impaired automatization of balance, and in time estimation, a non-motor cerebellar task. Brain imaging studies have also shown anatomical, metabolic and activation differences in the cerebellum of dyslexics.37 The magnocellular theory There is a unifying theory that attempts to integrate all the findings mentioned above. A generalization of the visual theory, the magnocellular theory postulates that the magnocellular dysfunction is not restricted to the visual pathways but is generalized to all modalities visual and auditory as well as tactile. Furthermore, as the cerebellum receives massive input from various magnocellular systems in the brain, it is also predicted to be affected by the general magnocellular defect Stein et al., 2001. Through a single biological cause, this theory therefore manages to account for all known manifestations of dyslexia: visual, auditory, tactile, motor and, consequently, phonological. Beyond the evidence pertaining to each of the theories described previously, evidence specifically relevant to the magnocellular theory includes magnocellular abnormalities in the medial as well as the lateral geniculate nucleus of dyslexics' brains, poor performance of dyslexics in the tactile domain, and the co-occurrence of visual and auditory problems in certain dyslexics.37 Perceptual visual-noise exclusion hypothesis The concept of a perceptual noise exclusion Visual-Noise deficit is an emerging hypothesis, supported by research showing that dyslexic subjects experience difficulty in performing visual tasks such as motion detection in the presence of perceptual distractions, but do not show the same impairment when the distracting factors are removed in an experimental setting.39 The researchers have analogized their findings concerning visual discrimination tasks to findings in other research related to auditory discrimination tasks. They assert that dyslexic symptoms arise because of an impaired ability to filter out both visual and auditory distractions, and to categorize information so as to distinguish the important sensory data from the irrelevant.40 Research using functional brain scan technology Please help improve this section by expanding it. Further information might be found on the talk page or at requests for expansion. May 2008 A University of Hong Kong study argues that dyslexia affects different structural parts of children's brains depending on the language which the children read.41 The study focused on comparing children that were raised reading English and children raised reading Chinese. Using MRI technology researchers found that the children reading English used a different part of the brain than those reading Chinese. Researchers were surprised by this discovery and hope that the findings will help lead them to any neurobiological cause for dyslexia.41 Research also indicates Genetic factors Studies have linked several forms of dyslexia to genetic markers.424344 One major genetic study identified a region on chromosome 6, DCDC2, as possibly linked to dyslexia.7 As of 2007, genetic research in families with dyslexia have identified nine chromosome regions that may be associated with susceptibility to dyslexia. However, several of the major studies have not been replicated.45 Physiology Modern neuroimaging techniques such as functional Magnetic Resonance Imaging fMRI and Positron Emission Tomography PET have produced clear evidence of structural differences in the brains of children with reading difficulties. It has been found that people with dyslexia have a deficit in parts of the left hemisphere of the brain involved in reading, which includes the inferior frontal gyrus, inferior parietal lobule, and middle and ventral temporal cortex.46 47 Scientific studies of brains donated to medical research have revealed that there are anatomical differences in two parts of the dyslexic brain: the cerebral cortex and the thalamus. In 1979 Albert Galaburda of Harvard Medical School noticed anatomical differences in the language center in a dyslexic brain, showing microscopic differences known as ectopias and microgyria. Both affect the typical six-layer structure of the cortex. An ectopia is a collection of neurons that have pushed up from the lower layers of the cortex into the outermost one. A microgyrus is an area of cortex that includes only four layers instead of six. These differences affect connectivity and functionality of the cortex in critical areas related to auditory processing and visual processing, which seems consistent with the hypothesis that dyslexia stems from a phonological awareness deficit. Studies of both autopsied brains and living brains using neuroimaging techniques have shown that the brains of dyslexic children are symmetrical, unlike the asymmetrical brains of non-dyslexic readers who had larger left hemispheres.48 Scientists do not claim that all people with dyslexia have these structural brain differences. However, the studies are evidence that some children's reading problems are brain based. The challenge for researchers is to determine how these structural differences affect reading acquisition. 49 Effect of language orthography Some studies have concluded that speakers of languages whose orthography has a highly consistent correspondence between letter and sound e.g., Italian suffer less from the effects of dyslexia than speakers of languages where the letter-sound correspondence is less consistent e.g. English and French.50 In one of these studies, reported in Seymour et al.,51 the word-reading accuracy of first-grade children of different European languages was measured. English children had an accuracy of just 40%, whereas among children of most other European languages accuracy was about 95%, with French and Danish children somewhere in the middle at about 75%; Danish and French are known to have an irregular pronunciation. However, this does not mean that dyslexia is caused by orthography: instead, Ziegler et al.52 claim that the dyslexia suffered by German or Italian dyslexics is of the same kind as the one suffered by the English ones, supporting the theory that the origin of dyslexia is biological. In a study by Paulescu et al. Science, 2001 English, French, and Italian dyslexics were found to have the same brain function signature when studied with functional magnetic resonance imaging fMRI, a signature that differed from non-dyslexic readers. However, dyslexia has more pronounced effects on orthographically difficult languages, e.g., dyslexics have more difficulty in English than Italian. Modern theories of some forms of dyslexia uses orthography to test a hypothesis of psychological causation Characteristics Formal diagnosis of dyslexia is made by a qualified professional, such as a neurologist or an educational psychologist. Evaluation generally includes testing of reading ability together with measures of underlying skills such as tests of rapid naming, to evaluate short term memory and sequencing skills, and nonword reading to evaluate phonological coding skills. Evaluation will usually also include an IQ test to establish a profile of learning strengths and weaknesses. However, the use of a discrepancy between full scale IQ and reading level as a factor in diagnosis has been discred by recent research.53 It often includes interdisciplinary testing to exclude other possible causes for reading difficulties, such as a more generalized cognitive impairment or physical causes such as problems with vision or hearing. Recent advances in neuroimaging and genetics provide evidence that could potentially help identify children with dyslexia before they learn to read in the future. However, such tests have not yet been developed and more research is needed before such testing could be considered reliable. Speech, hearing and listening Speech delays may be an early warning sign of dyslexia. Many dyslexics may have problems processing and decoding auditory input prior to reproducing their own version of speech. Early stuttering or cluttering can also be warning signs of dyslexia. Many dyslexics also can have problems with speaking clearly. They can mix up sounds in multi-syllabic words ex: aminal for animal, bisghetti for spaghetti, hekalopter for helicopter, hangaberg for hamburger, mazageen for magazine, etc. They also can have problems speaking in full sentences. They can have trouble correctly articulating Rs and Ls as well as Ms and Ns. They often have immature speech. They may still be saying wed and gween instead of red and green in second or third grade. Many dyslexics might have speech therapy in special education. They may have fast speech, cluttered speech, or hesitant speech. 54 55 Reading requires the sounding out of words. Therefore, it makes sense that children with speech problems can end up having reading problems later. Many have problems with speech due to problems with auditory processing disorder issues. Many dyslexics have problems with phonemic awareness. Phonemes are the smallest units in spoken language. The Auditory related underlying causes of dyslexia may be partially remediated by auditory therapy or speech therapy, which help with phonemic awareness. This may help to make sense of phonics which may help with phonological awareness, which is needed to sound out words. Many acquire auditory processing disorder as an underlying cause of dyslexia from glue ear, otitis media. Some shared symptoms of the speech/hearing deficits and dyslexia: Confusion with before/after, right/left, and so on Difficulty learning the alphabet Difficulty with word retrieval or naming problems Difficulty identifying or generating rhyming words, or counting syllables in words phonological awareness Difficulty with hearing and manipulating sounds in words phonemic awareness Difficulty distinguishing different sounds in words auditory discrimination Difficulty in learning the sounds of letters Difficulty associating individual words with their correct meanings Difficulty with time keeping and concept of time Confusion with combinations of words Due to fear of speaking incorrectly, some children become withdrawn and shy or become bullies out of their inability to understand the social cues in their environment Reading and spelling Spelling errors - Because of difficulty learning letter-sound correspondences, individuals with dyslexia might tend to misspell words, or leave vowels out of words. Letter order - Dyslexics may also reverse the order of two letters especially when the final, incorrect, word looks similar to the intended word e.g., spelling dose instead of does. Highly phoneticized spelling - Dyslexics also commonly spell words inconsistently, but in a highly phonetic form such as writing shud for should. Dyslexic individuals also typically have difficulty distinguishing among homophones such as their and there. Reading - Due to dyslexics' excellent long term memory, young students tend to memorize beginning readers, but are unable to read individual words or phrases. Vocabulary - Having a small written vocabulary, even if they have a large spoken vocabulary. Writing and motor skills Because of literacy problems, an individual with dyslexia may have difficulty with handwriting. This can involve slower writing speed than average or poor handwriting characterised by irregularly formed letters. They may use inappropriate words when writing. Some studies have also reported gross motor difficulties in dyslexia, including motor skills disorder. This difficulty is indicated by clumsiness and poor coordination. The relationship between motor skills and reading difficulties is poorly understood but could be linked to the role of the cerebellum and inner ear in the development of reading and motor abilities.56 Mathematical abilities Dyslexia should not be confused with dyscalculia, a learning disability marked by severe difficulties with mathematics. Individuals with dyslexia can be gifted in mathematics while having poor reading skills. However, in spite of this they might have difficulty with word problems i.e., descriptive mathematics, engineering, or physics problems that rely on written text rather than numbers or formulas. Individuals with dyslexia may also have difficulty remembering mathematical facts, such as multiplication tables, learning the sequence of steps when performing calculations, such as long division, and other mathematics which involve remembering the order in which numbers appear. This may be exhibited by having a slow response in mathematical drills and difficulty with word problems. Creativity, arts, business A study has found that entrepreneurs are five times more likely to suffer from dyslexia than average citizens57. Management See also: Dyslexia treatment There is no cure for dyslexia, but dyslexic individuals can learn to read and write with appropriate education or treatment. There is wide research evidence indicating that specialized phonics instruction can help remediate the reading deficits. The fundamental aim is to make children aware of correspondences between graphemes and phonemes, and to relate these to reading and spelling. It has been found that training, that is also focused towards visual language and orthographic issues, yields longer-lasting gains than mere oral phonological training.29 Teachers are also using audiobooks as a way of teaching textbooks in an engaging way to those with dyslexia. In the UK, one of the biggest charities is Listening Books, offering members a streaming service over the internet. www.listening-books.org.uk Effective training is given by teachers at school or kindergarten. Meta-analysis evaluating the effects of phonological awareness instruction has shown that word reading skills of all children, those with a risk for reading problems as well as those developing typically, improved their reading in systematic phonics instruction, a method that encourages a word to be recognised through the building of its constituent sounds. Basic phonemic awareness instruction did not, however, improve spelling in disabled readers.58 None of the studies included measures of reading fluency.59 The core deficit of dyslexia is in learning to read at the word level 60, and individuals with dyslexia require more practice to master skills in their areas of deficit. In the circumstances where typically developing children need 30 to 60 hours training, the number of hours that has resulted in optimistic conclusions concerning the treatability of dyslexia is between 80 and 100 hours, or less if the intervention is started sufficiently early. Only approximately 20% of adults with early reading difficulties have acquired fluent reading skills in adulthood. 29 Functional MRI fMRI studies have shown changes in the brains of dyslexic children and adults with phonics tutoring, along with improved performance on tests of phonemic awareness and text decoding.61 62 Functional MRI studies have also shown changes in the brain and spelling improvement of dyslexic children taught spelling phonetically in an orthographic manner. 63 One factor that characterises the field of dyslexia treatment is the incessant flow of alternative therapies for developmental and learning disabilities. These controversial treatments include nutritional supplements, special diets, homeopathy, and osteopathy/chiropractic manipulation.64 Facts and statistics In the United States, researchers estimate the prevalence of dyslexia to range from five to nine percent of school-aged children, though some have put the figure as high as 17 percent.6566 Recent studies indicate that dyslexia is particularly prevalent among small business owners, with roughly 20 to 35 percent of U. S. and British entrepreneurs being affected. Researchers consider that many dyslexic entrepreneurs are successful by delegating responsibilities and excelling at verbal communication.67 Dyslexia's main manifestation is a difficulty in developing word-level reading skills in elementary school children. Those difficulties result from reduced ability to associate visual symbols with verbal sounds. While motivational factors must also be reviewed in assessing poor performance, dyslexia is considered to be developmental. Most scientific criteria for dyslexia exclude cases that can be explained as arising from environmental factors such as lack of education or total sensory deficits. Dyslexia can be substantially compensated for with proper therapy, training, and assistive technology. Many coping strategies are developed subconsciously by the individual dyslexic. Dyslexia has many variations dependent on the cultural choice of visual notation of speech. So the nature of the notation used in different cultures creates different types of problems for their groups of dyslexics. The differences between the English text and Chinese characters is a good example. Legal and educational support issues In the United States, Canada, New Zealand and in the United Kingdom, it has been claimed that there is a lack of adequate support and a general lack of interest in the learning disabilities of children in public schools.citation needed This has recently led to legal action by private parties against public schools in the United States and state schools in the United Kingdom. In English law, the recent case of Skipper v Calderdale Metropolitan Borough School 2006 EWCA Civ 238 the Court of Appeal applied Phelps v London Borough of Hillingdon 2001 2 AC 619 as the landmark case on the failure to diagnose dyslexia see duty of care in English law, and to hold that the appellant could pursue her claim against her school for humiliation, lost confidence, and lost self-esteem, and for loss of earnings following its failing to diagnose and treat her dyslexia despite the fact that, as Latham LJ. says at para 29: The extent to which her dyslexia could have been ameliorated or provided for will always remain uncertain, as will the extent to which that would have affected her performance in public examinations; the evidence that we have includes material to suggest that she, not surprisingly, reacted adversely to the break-up of her parents marriage when she was 15, in other words at a critical time in her education. Whether any improvement in her examination results would have led to her life taking a significantly different course will also be a matter for some speculation. Some charitable organizations like the Scottish Rite Foundation have undertaken the task of testing for dyslexia and making training classes and materials available, often without cost, for teachers and students.686970 In England and Wales, the failure of schools to diagnose and provide remedial help for dyslexia following the House of Lords decision in the case of Pamela Phelps has created an entitlement for students with dyslexia in Higher education to receive support funded via the Disabled Students Allowance. Support can take the form of IT equipment software and hardware as well as personal assistance, also known as non-medical helper support. Dyslexic students will also be entitled to special provision in examinations such as additional time to allow them to read and comprehend exam questions. The British Disability Discrimination Act 1995 also covers dyslexia. In some cases, people have 'coping strategies' which cease to work in certain circumstances for example, where someone who stutters or has dyslexia is placed under stress. If it is possible that a person's ability to manage the effects of the impairment will break down so that these effects will sometimes occur, this possibility must be taken into account when assessing the effects of the impairment. - Paragraph A8, Guidance to the Definitions of Disability Controversy Some disagreement exists as to whether dyslexia does indeed exist as a condition, or whether it simply reflects individual differences among different readers. The Dyslexia Myth is a documentary that appeared as part of the Dispatches series produced by British broadcaster Channel 4.71 First aired in September 2005, it claims to expose myths and misconceptions that surround dyslexia. It argues that the common understanding of dyslexia is not only false but makes it more difficult to provide the reading help that hundreds of thousands of children desperately need. Drawing on years of intensive academic research on both sides of the Atlantic, it challenged the existence of dyslexia as a separate condition, and highlighted the many different forms of reading style. The documentary only focused on the reading difficulties that dyslexics encounter. As discussed in previous headings, dyslexia is more than a mere reading disability, and commonly includes symptoms that extend beyond reading difficulties. However, these symptoms are not included in the DSM-IV list of symptoms by which Reading Disorder is diagnosed USA. Julian Elliott, a psychologist at Durham University in the UK, disputes the characterization of dyslexia as a medical condition, and believes it should be treated simply as a reading difficulty. According to Elliot, parents don't want their child to be considered lazy, thick or stupid. If they get called this medically diagnosed term, dyslexic, then it is a signal to all that it's not to do with intelligence.72 Elliot believes that children of all levels of intelligence may struggle with learning to read, and that all can be helped by educational strategies appropriate to their needs. He feels that resources are wasted on diagnosis and testing, and favors early intervention programs for all struggling readers.73 However, John Everatt of the University of Surrey, also in the UK, has demonstrated that dyslexic students can be distinguished from other children with low reading achievement by testing geared to assessing their strengths as well as weaknesses. Dyslexic children tend to score significantly better than other children, including non-impaired children, on tests of creativity, spatial memory, and spatial reasoning. Dyslexic children also perform better than other reading-impaired children on tests of vocabulary and listening comprehension. Everatt suggests that dyslexic children may be better served by educational intervention which includes strategies geared to their unique strengths in addition to skill remediation, and thus recommends more comprehensive evaluation and targeted interventions. 74 Dyslexia in literature, film, and television Main article: Dyslexia in fiction Dyslexic characters have featured in a number of works of fiction. Notable works include Aamir Khan's 2007 award - winning film Taare Zameen Par, Henry Winkler's Hank Zipzer series of children's books, and Jennifer Weiner's 2002 novel, In Her Shoes which was adapted as the 2005 film, In Her Shoes. The Simpsons character Duffman is dyslexic, according to the 19th season episode Eternal Moonshine of the Simpson Mind. See also Foggy brain Child development References ^ Stanovich, KE. 1988 Explaining the differences between the dyslexic and the garden-variety poor reader: the phonological-core variable-difference model. Journal of Learning Disabilities, 2110:590-604. ^ A Conversation with Sally Shaywitz, M.D., author of Overcoming Dyslexia 2007-10-15. Retrieved on 2008-04-21. ^ Murphy, Martin F 2004. Dyslexia, An Explanation. Flyleaf Press. ISBN. ^ Deborah Moncrieff 2004-04-12. Temporal Processing Deficits in Children with Dyslexia. Audiology Online. ^ Dalby, JT 1985. Taxonomic separation of attention deficit disorders and developmental reading disorders.. Contemporary Educational Psychology 10: 228-234. doi:10.1016/0361-476X8590019-0. ^ Cope, N; Harold D, Hill G, Moskvina V, Stevenson J, Holmans P, Owen MJ, O'Donovan MC, Williams J April 2005. Strong evidence that KIAA0319 on chromosome 6p is a susceptibility gene for developmental Dyslexia. American Journal of Human Genetics 76 4: 581-91. doi:10.1086/429131. ^ a b Meng, H; Smith SD, Hager K, Held M, Liu J, Olson RK, Pennington BF, DeFries JC, Gelernter J, O'Reilly-Pol T, Somlo S, Skudlarski P, Shaywitz SE, Shaywitz BA, Marchione K, Wang Y, Paramasivam M, LoTurco JJ, Page GP, Gruen JR November 22 2005. DCDC2 is associated with reading disability and modulates neuronal development in the brain. Proceedings of the National Academy of Sciences 102 47: 17053-8. doi:10.1073/pnas.0508591102. PMID 16278297. ^ Schumacher, J; Anthoni H, Dahdouh F, Konig IR, Hillmer AM, Kluck N, Manthey M, Plume E, Warnke A, Remschmidt H, Hulsmann J, Cichon S, Lindgren CM, Propping P, Zucchelli M, Ziegler A, Peyrard-Janvid M, Schulte-Korne G, Nothen MM, Kere J Jan 2006. Strong genetic evidence of DCDC2 as a susceptibility gene for Dyslexia. American Journal of Human Genetics 78 1: 52-62. doi:10.1086/498992. ^ Lyon, G.Reid; Fletcher, J.M.; Shaywitz, S.E.; Shaywitz, B.A.; Torgesen, J.K.; Wood, F.B.; Schulte, A.; Olson, R. 2001. Rethinking learning disabilities. Rethinking special education for a new century: 259-287. Retrieved on 2007-06-11. ^ Clark, Diana B, et.al. 1995. Dyslexia: Theory and Practice of Remedial Instruction. York Press. ^ National Institute of Child Health and Human Development, 2000; Snow et al., 1998; Torgesen, 2000. Retrieved from http://www.ldonline.org/article/14907 October 16, 2007. ^ Fink, Rosalie 2006. Why Jane and John Couldn't Read--And How They Learned: A New Look at Striving Readers. International Reading Association. ISBN 978-0872075924. ^ BERKHAN O. Neur. Zent 28 1917 ^ Uber Dyslexie . Archiv fur Psychiatrie 15: 276-278. ^ Snowling, Margaret J. 1996-11-02. Dyslexia: a hundred years on. BMJ 313 7065: 1096. PMID 8916687. Retrieved on 2007-06-08. ^ Hinshelwood, J. 1917. Congenital Word-blindness. HK Lewis \ Co., ltd.. ^ Orton, ST 2519. 'Word-blindness' in school children.. Archives of Neurology and Psychiatry 14: 285-516. ^ Henry, MK 1998. Structured, sequential, multisensory teaching: The Perlow legacy. Annals of Dyslexia 48: 1. doi:10.1007/s11881-998-0002-9. ^ Orton, S.T. 1928. Specific reading disability-strephosymbolia. Journal of the American Medical Association 90 14: 1095-1099. ^ Geschwind, N 1982. Biological associations of left-handedness. Annals of Dyslexia 33: 29-40. doi:10.1007/BF02647994. ^ Galaburda, A.M.; Menard, M.T.; Rosen, G.D. 1994-08-16. Evidence for Aberrant Auditory Anatomy in Developmental Dyslexia. Proceedings of the National Academy of Sciences 91 17: 8010-8013. doi:10.1073/pnas.91.17.8013 inactive 2008-06-25. PMID 8058748. Retrieved on 2007-06-17. ^ Was Orton Right? New Study Examines How The Brain Works In Reading; Offers Key To Better Understanding Dyslexia. Science Daily 2003-05-19. Retrieved on 2007-06-17. ^ Turkeltaub, P.E.; Gareau, L.; Flowers, D.L.; Zeffiro, T.A.; Eden, G.F. 2003. Development of neural mechanisms for reading dead link - Scholar search. Nature Neuroscience 6 7: 767-773. Retrieved on 2007-06-17. ^ Orton, Samuel. Word Blindness in School Children. Archives or Neurology and Psychiatry 14:285-516. ^ Goeke, Jennifer 2006. Orton-Gillingham and Orton-Gillingham-based reading instruction: a review of the literature. Journal of Special Education. ^ Bradley, L; Bryant, PE 1983. Categorizing sounds and learning to read: A Causal connection.. Nature 30 2: 419-421. doi:10.1038/301419a0. ^ Alexander, A; Anderson, H, Heilman, P, Voeller, K, Torgesen, J 1991. Phonological awareness training and the remediation of analytic decoding deficits in a group of severe dyslexics. Annals of Dyslexia 41: 193-206. doi:10.1007/BF02648086. ^ Collins, David and Rourke, Byron October 2003. taylorandfrancis.metapress.com/index/H47FWM65HHJAP0K6.pdf Learning-disabled Brains: A Review of the Literature. Journal of Clinical and Experimental Neuropsychology 25 7: 1011-1034. Retrieved on 2007-07-11. ^ a b c Lyytinen, Heikki, Erskine, Jane, Aro, Mikko, Richardson, Ulla 2007, Reading and reading disorders, in Hoff, Erika, Blackwell Handbook of Language Development, Blackwell, pp. 454-474, ISBN 9781405132534 ^ a b c d e f Castles, A., Coltheart, M. 1993. Varieties of developmental dyslexia. Cognition, 472, 149-180 ^ Castles, A., Coltheart, M. 1993. Varieties of developmental dyslexia. Cognition, 472, 149-180 ^ Manis, F., Seidenberg, M., Doi, L., McBride-Chang, C., Petersen, A., 1996. On the basis of two subtypes of developmental dyslexia. Cognition, 59, 157-195 ^ Wolf, M., Bowers, P. G., 1999. The double-deficit hypothesis for the developmental dyslexias. Journal of educational psychology, 91, 415-438 ^ Cluttering as a Complex of Learning Disabilities ^ Dalby, J.T. 1986. An ultimate view of reading ability. International Journal of Neuroscience 30 3: 227-230. Gordon and Breach, Science Publishers, Inc.. PMID 3759349. ^ Kailing, Timothy D. 2008. Native Reading. Elliptical Research Books. ISBN 978-1434848819. ^ a b c d e Ramus, Franck; Stuart Rosen, Steven C. Dakin, Brian L. Day, Juan M. Castellote, Sarah White and Uta Frith April 2003. Theories of developmental dyslexia: insights from a multiple case study of dyslexic adults. Brain 126 4: 841-865. Oxford University Press. doi:10.1093/brain/awg076. PMID 12615643. Retrieved on 2007-05-27. ^ Wright, Beverlyqwefojq wefi o this is a stupid site.; Linda J. Lombardino, Wayne M. King, Cynthia S. Puranik, Christiana M. Leonard and Michael M. Merzenich 1997. Deficits in auditory temporal and spectral resolution in language-impaired children. Nature 387: 176-8. doi:10.1038/387176a0. ^ Sperling, Anne J.; Zhong-Lin Lu, Franklin R. Manis, Mark S. Seidenberg 2006. Motion-Perception Deficits and Reading Impairment: It's the Noise, Not the Motion. Psychological Science 17 12: 1047-1053. Association for Psychological Science. doi:10.1111/j.1467-9280.2006.01825.x. ^ Sperling, Anne J.; Lu, Z.L.; Manis, F.R.; Seidenberg, M.S. 2005. Deficits in perceptual noise exclusion in developmental dyslexia. Nature Neuroscience 8: 862-863. doi:10.1038/nn1474. ISSN 1097-6256. Retrieved on 2007-06-12. ^ a b Study: Dyslexia differs by language. PsycPORT 8 April 2008. Retrieved on 2008-04-26. ^ Grigorenko, EL Jan 2001. Developmental dyslexia: an update on genes, brains, and environments. Journal of child psychology and psychiatry, and allied disciplines 42 1: 91-125. doi:10.1017/S0021963001006564. ^ Grigorenko, EL; Wood FB, Meyer MS, Hart LA, Speed WC, Shuster A, Pauls DL Jan 1997. Susceptibility loci for distinct components of developmental dyslexia on chromosomes 6 and 15. American journal of human genetics 60 1: 27-39. ^ Grigorenko, EL; Wood FB, Meyer MS, Pauls DL Feb 2000. Chromosome 6p influences on different dyslexia-related cognitive processes: further confirmation. American journal of human genetics 66 2: 715-23. doi:10.1086/302755. ^ Schumacher, Johannes; Per Hoffmann, Christine Schmäl, Gerd Schulte-Körne and Markus M Nöthen 16 February 2007. Genetics of dyslexia: the evolving landscape. Journal of Medical Genetics 44: 289-297. BMJ Publishing Group. doi:10.1136/jmg.2006.046516. PMID 17307837. Retrieved on 2007-05-27. ^ Fan Cao, Tali Bitan, Tai-Li Chou, Douglas D. Burman, James R. Booth 2006. Deficient orthographic and phonological representations in children with dyslexia revealed by brain activation patterns. Journal of Child Psychology and Psychiatry November 2006 47: 1041-1050. doi:10.1111/j.1469-7610.2006.01684.x. Retrieved on 2007-10-06. ^ Shaywitz BA, Lyon GR, Shaywitz SE 2006. The role of functional magnetic resonance imaging in understanding reading and dyslexia.. PubMed 2006 47: 1041. doi:10.1207/s15326942dn3001_5. PMID 16925477. Retrieved on 2007-10-06. ^ Haslam, R, Dalby, J.T., Johns, RD and Rademaker,A.W. 1981. Cerebral asymmetry in developmental dyslexia. Archives of Neurology 27: 23-25. PMID 7305694. ^ Sherman, Gordon. Can Neuroscience Help to Demystify Dyslexia? Schwab Learning. Retrieved from http://www.schwablearning.org/articles.aspx?r=430 October 8, 2007. ^ Scientists Say Severity of Dyslexia Depends on Language, The Tech News Briefs, Los Angeles Times 2001-03-16. Retrieved on 2006-06-06. ^ Seymour, P. H. K., Aro, M., Erskine, J. M. 2003. Foundation literacy acquisition in European orthographies. British Journal of Psychology, 94, 143 - 174. ^ Johannes C. Ziegler, Conrad Perry, Anna Ma-Wyatt, Diana Ladner, and Gerd Schulte-Körne, Developmental dyslexia in different languages: Language-specific or universal? Journal of Experimental Child Psychology 169 - 193 ^ Fletcher first=Jack M; Francis DJ, Rourke BP, Shaywitz SE, Shaywitz BA. November 1992. The validity of discrepancy-based definitions of reading disabilities.. J Learn Disabil 259:555-61, 573. 25 9: 555-61, 573. ISSN ISSN-0022-2194. Retrieved on 2007-06-15. ^ Stephen Wilcox - Dyslexia Vision ^ This book is written in plain language to make it easier to read for student with dyslexia as well as busy teachers and ^ Nicolson, R. and Fawcett, A. Nov 1999. Developmental dyslexia: the role of the cerebellum. Dyslexia: An International Journal of Research and Practice 5: 155-7. ^ cass.city.ac.uk Entrepreneurs five times more likely to suffer from dyslexia ^ Ehri, Nunes, Willows, Schuster, Yaghoub-Zadeh, Shananan 2001. Phonemic Awareness Instruction Helps Children Learn to Read: Evidence From the National Reading Panel's Meta-Analysis. Reading Research Quarterly 36 3: 250-287. doi:10.1598/RRQ.37.2.1. Retrieved on 2007-07-31. ^ Necoechea, D. M. 2003. The role of reading intervention research in the identification of children with reading difficulties: A meta-analysis of the literature funded by the NICHD.. Advances in Learning and Behavioral Disabilities 16: 83-161. doi:10.1016/S0735-004X0316004-1. ^ Clark, Diana B, et.al. 2005. Dyslexia: Theory and Practice of Remedial Instruction. York Press. ^ Shaywitz, B.; Shaywitz, S.; Blachman, B.; Pugh, K.R.; Fulbright, R.; Skudlarski, P.; Others, 2004. Development of left occipitotemporal systems for skilled reading following a phonologically based intervention in children. Biological Psychiatry 55: 926-933. doi:10.1016/j.biopsych.2003.12.019. ^ Eden, G.F.; Jones, K.M.; Cappell, K.; Gareau, L.; Wood, F.B.; Zeffiro, T.A.; Dietz, N.A.E.; Agnew, J.A.; Flowers, D.L. 2004. Neural Changes following Remediation in Adult Developmental Dyslexia. Neuron 44 3: 411-422. doi:10.1016/j.neuron.2004.10.019. Retrieved on 2007-07-18. ^ Dahms, Joel. 2006. Spelling Out Dyslexia. Northwest Science Technology. ^ Bull L 2008. Survey of complementary and alternative therapies used by children with specific learning difficulties dyslexia. Int J Lang Commun Disord. doi:10.1080/13682820802015643. PMID 18608596. ^ Shaywitz, Sally E.; Bennett A. Shaywitz August 2001. The Neurobiology of Reading and Dyslexia. Focus on Basics 5 A. National Center for the Study of Adult Learning and Literacy. ^ Learning Disabilities: Multidisciplinary Research Centers, NIH Guide, Volume 23, Number 37, October 21, 1994, Full Text HD-95-005 LDRC longitudinal, epidemiological studies show that RD dyslexia affect at least 10 million children, or approximately 1 child in 5. ^ Brent Bowers 2007-12-06. Tracing Business Acumen to Dyslexia, New York Times. Cites a study by Julie Logan, professor of entrepreneurship at Cass Business School in London, among other literature. ^ Dyslexia Workshop Topics. Alabama Scottish Rite Foundation Learning Centers. Retrieved on June 6, 2006. ^ Scottish Rite Masonic Children's Learning Centers. Retrieved on June 6, 2006. ^ Scottish Rite Philanthropy. The Scottish Rite Bodies of Austin. Retrieved on June 6, 2006. ^ The Dyslexia Myth. Dispatches. Channel 4. ^ Blair, Alexandra 2007-05-28. Dyslexia 'is used by parents as excuse for slow children'', Times Newspapers LtdUnited Kingdom. Retrieved on 2007-05-29. ^ Is dyslexia just a myth?, Guardian Unlimited 2005-09-07. Retrieved on 2007-05-29. ^ Everatt, John; Weeks, Sally; Brooks, Peter 23 Jul 2007 online. Profiles of Strengths and Weaknesses in Dyslexia and Other Learning Difficulties. Dyslexia In Press: 16. John Wiley Sons. doi:10.1002/dys.342. Retrieved on 2007-07-26. 26 TINKER M. A. and GOODENOUGH F.L. Mirror reading as a method of analysing factors involved in word perception J. Educ. Psych. 1931, 22, 493 External links Historical Samuel T Orton and June L Orton Personal Papers and Manuscripts at the Columbia University Health Science Library collection Research papers, articles and media Auditory and visual automatic attention deficits in developmental dyslexia Dyslexia - A common learning disability Auditory and Visual Dynamic Processing: Separate Influences in Reading? Diagnosing dyslexia in children - a guide for parents Auditory frequency discrimination in adult developmental dyslexics Can sensitivity to auditory frequency modulation predict children's phonological and reading skills? Children of the Code - A social-education Project about the code and the challenge of learning to read it Dynamic sensory sensitivity and children's word decoding skills Dyslexia Unit Publications 1981-2003 list of research papers which can be located at academic publishers or Googled Effect of intensive training on auditory processing and reading skills The Neural Basis of Reading articles 1995 - 2005 On the Relationship between Dynamic Visual and Auditory Processing and Literacy Skills; Results from a Large Primary-school Study Temporal Processing Deficits in Children with Dyslexia Haskins Laboratories reading research Regional associations and organizations Adult Dyslexia Organisation UK British Dyslexia Association Dyslexia Action UK Canadian Dyslexia Association International Dyslexia Association US Dyslexia International - Tools and Technologies DITT The Dyslexia Guide, Dyslexia Ayrshire Scotland, A to Z and FAQ Disabled Law Students Association LDLSA Dyslexia Association of Singapore SG Dyslexia Association of Ireland Support groups and organizations LD Online A service of public television station WETA in Washington, D.C., in association the National Joint Committee on Learning Disabilities US Help for Parents of Dyslexic Children Dyslexia Correction Information 'The Mathematician Who Can't Add Up' Emma King Helping Children with Learning Disability in India Freeview 'Snapshot' video by the Vega Science Trust Dyslexia, Learning Disabilities, and Special Education Information Ghotit dyslexia spelling, a free online LD assisting resource. Improving instruction for students with Dyslexia. Dyslexic People Overcoming Dyslexia Article in 'fortune' magazine about how dyslexia affects Richard Branson and Charles Schwab v d e WHO ICD-10 mental and behavioral disorders F · 290-319 Neurological/symptomatic Dementia Alzheimer's disease, multi-infarct dementia, Pick's disease, Creutzfeldt-Jakob disease, Huntington's disease, Parkinson's disease, AIDS dementia complex, Frontotemporal dementia, Elopement, Sundowning, Wandering · Delirium · Post-concussion syndrome · Organic brain syndrome Psychoactive substance alcohol drunkenness, alcohol dependence, alcoholic hallucinosis, Alcohol withdrawal, delirium tremens, Korsakoff's syndrome, alcohol abuse · opioids opioid dependency · sedative/hypnotic benzodiazepine withdrawal · cocaine cocaine dependence · general Intoxication, Drug abuse, Physical dependence, Withdrawal Psychotic disorder Schizophrenia disorganized schizophrenia · Schizophreniform disorder · Schizotypal personality disorder · Delusional disorder · Folie à deux · Schizoaffective disorder Mood affective Mania · Bipolar disorder · Clinical depression · Cyclothymia · Dysthymia Neurotic, stress-related and somatoform Anxiety disorder Agoraphobia, Panic disorder, Panic attack, Generalized anxiety disorder, Social anxiety, Social phobia · OCD · Acute stress reaction · PTSD · Adjustment disorder · Conversion disorder Ganser syndrome · Somatoform disorder Somatization disorder, Body dysmorphic disorder, Hypochondriasis, Nosophobia, Da Costa's syndrome, Psychalgia · Neurasthenia Physiological/physical behavioral Eating disorder: Anorexia nervosa · Bulimia nervosa Sleep disorder: Dyssomnia Hypersomnia, Insomnia · Parasomnia REM behavior disorder, Night terror · Nightmare Sexual dysfunction: Erectile dysfunction · Premature ejaculation · Vaginismus · Dyspareunia · Hypersexuality · Female sexual arousal disorder Postpartum depression · Postnatal psychosis Adult personality and behavior Personality disorder · Passive-aggressive behavior · Kleptomania · Trichotillomania · Voyeurism · Factitious disorder · Munchausen syndrome · Ego-dystonic sexual orientation · Fetishism Mental retardation Mental retardation Psychological development developmental disorder Specific: speech and language expressive language disorder, aphasia, expressive aphasia, receptive aphasia, Landau-Kleffner syndrome, lisp · Scholastic skills dyslexia, dysgraphia, Gerstmann syndrome · Motor function developmental dyspraxia Pervasive: Autism · Rett syndrome · Asperger syndrome Behavioral and emotional, childhood and adolescence onset ADHD · Conduct disorder · Oppositional defiant disorder · Separation anxiety disorder · Selective mutism · Reactive attachment disorder · Tic disorder · Tourette syndrome · Speech stuttering · cluttering v d e Symptoms and signs: Speech and voice R47-R49, 784 Aphasia/Dysphasia Expressive aphasia - Receptive aphasia - Conduction aphasia Other speech disturbances Dysarthria - Schizophasia Symbolic dysfunctions Dyslexia - Alexia - Agnosia Prosopagnosia, Astereognosis - Apraxia Ideomotor apraxia - Acalculia - Agraphia Voice disturbances Dysphonia - Aphonia Retrieved from http://en..org/wiki/Dyslexia Categories: Dyslexia | Disability | Neurology | Special education | People with dyslexiaHidden categories: Pages with DOIs broken since 2008 | All articles with dead external links | Articles with dead external links since June 2008 | Articles containing Ancient Greek language text | Articles to be expanded since May 2008 | All articles to be expanded | All articles with statements | Articles with statements since May 2008 Views Article Discussion this page History Personal tools Log in / create account Navigation Main page Contents Featured content Current events Random article Search Go Search Interaction Community portal Recent changes Contact Donate to Help Toolbox What links here Related changes Upload file Special pages Printable version Permanent link Cite this page Languages Afrikaans Català Česky Cymraeg Dansk Deutsch Ελληνικά Español Esperanto Euskara Français Galego Ido Ã?slenska Italiano עברית LatvieÅ¡u Magyar Bahasa Melayu Nederlands 日本語 ‪Norsk bokmÃ¥l‬ Polski Português РуÑ?Ñ?кий Simple English SlovenÄ?ina СрпÑ?ки / Srpski Suomi Svenska Tiếng Việt Türkçe УкраїнÑ?ька ä¸æ–‡ This page was last modified on 28 August 2008, at 19:0
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